Myopathy / rhabdomyolysis
Rare cases of rhabdomyolysis with acute renal failure secondary to myoglobinuria have
been reported with rosuvastatin and with other drugs in this class.
Uncomplicated myalgia has been reported in rosuvastatin-treated patients (see ADVERSE
REACTIONS). Creatine kinase (CK) elevations (>10 times upper limit of normal) occurred in
0.2% to 0.4% of patients taking rosuvastatin at doses up to 40 mg in clinical studies. Treatment-related myopathy, defined as muscle aches or muscle weakness in conjunction with increases in
CK values >10 times upper limit of normal, was reported in up to 0.1% of patients taking
rosuvastatin doses of up to 40 mg in clinical studies. In clinical trials, the incidence of myopathy
and rhabdomyolysis increased at doses of rosuvastatin above the recommended dosage range (5
to 40 mg). In postmarketing experience, effects on skeletal muscle, e.g. uncomplicated myalgia,
myopathy and, rarely, rhabdomyolysis have been reported in patients treated with HMG-CoA
reductase inhibitors including rosuvastatin. As with other HMG-CoA reductase inhibitors,
reports of rhabdomyolysis with rosuvastatin are rare, but higher at the highest marketed dose (40
mg). Factors that may predispose patients to myopathy with HMG-CoA reductase inhibitors
include advanced age (≥65 years), hypothyroidism, and renal insufficiency.
Consequently:
1. Rosuvastatin should be prescribed with caution in patients with predisposing factors for
myopathy, such as, renal impairment (see DOSAGE AND ADMINISTRATION), advanced
age, and inadequately treated hypothyroidism.
2. Patients should be advised to promptly report unexplained muscle pain, tenderness, or
weakness, particularly if accompanied by malaise or fever. Rosuvastatin therapy should be
discontinued if markedly elevated CK levels occur or myopathy is diagnosed or suspected.
3. The 40 mg dose of rosuvastatin is reserved only for those patients who have not achieved their
LDL-C goal utilizing the 20 mg dose of rosuvastatin once daily (see DOSAGE AND
ADMINISTRATION).
4. The risk of myopathy during treatment with rosuvastatin may be increased with concurrent
administration of other lipid-lowering therapies or cyclosporine, (see CLINICAL
PHARMACOLOGY, Drug Interactions, PRECAUTIONS, Drug Interactions, and DOSAGE
AND ADMINISTRATION). The benefit of further alterations in lipid levels by the
combined use of rosuvastatin with fibrates or niacin should be carefully weighed against
the potential risks of this combination. Combination therapy with rosuvastatin and
gemfibrozil should generally be avoided. (See DOSAGE AND ADMINISTRATION and
PRECAUTIONS, Drug Interactions).
5.The risk of myopathy during treatment with rosuvastatin may be increased in
circumstances which
increase rosuvastatin
drug
levels
(see CLINICAL
PHARMACOLOGY, Special Populations, Race and Renal Insufficiency, and
PRECAUTIONS, General).
6.Rosuvastatin therapy should also be temporarily withheld in any patient with an acute,
serious condition suggestive of myopathy or predisposing to the development of renal
failure secondary to rhabdomyolysis (e.g., sepsis, hypotension, dehydration, major
surgery, trauma, severe metabolic, endocrine, and electrolyte disorders, or uncontrolled
seizures).